Hyperbaric Oxygen Treatments Reviews

Post Polio Syndrome

Post Polio Syndrome is the progressive deterioration of the nerves and the leg muscles which causes atrophy of the muscles and leads to the inability to walk unaided. It many times leads to loss of strength in the arms also. The person’s sense of balance usually becomes worse and they end up in a wheelchair. The progression of the polio type symptoms can happen 20 to 60 years after the original episode.

New research has discovered that the effects of Polio can be greatly reversed!

In the old days the medical establishment told patients with polio that a virus entered into the spinal fluid and attacked a nerve and killed the nerve. The patient was then told that the dead nerve would prevent the leg from further growth and development.

The truth is that the virus did enter into the spinal fluid but it did not kill the nerve. The nerve is still alive it is only dehydrated. The nerve can be rehydrated and brought back to life!

Here is how it happened.

There are two types of infection, bacterial and viral. Bacteria are larger and cannot get past the blood-brain barrier into the spinal fluid. Viruses are much smaller and are able to sneak past the blood-brain barrier into the spinal fluid and cause infections. Polio, meningitis, chicken pox, and the common cold are all viruses. The polio virus is able to get past the body’s immune system and into the spinal fluid and cause an inflammation of the meninges. This is where the term meningitis comes from. The spinal fluid is the same fluid that is removed when a spinal tap is done. There are three layers of the meninges. They are called the dura matter, pia matter, and the arachnoid matter. When a virus enters into the spinal fluid and causes inflammation then the meninges begins to swell. If the swelling or inflammation continues long enough then the meninges begin to touch and stick together like flypaper. This forms an adhesion. Adhesions form scar tissue which interferes with the proper flow of the spinal fluid. The fluid now has to go around the scar tissue. The area just the other side of the adhesion begins to dry out. That nerve will then become dehydrated. Wherever that nerve goes will now be affected. For instance if the dehydrated nerve goes to the stomach then ulcers can develop.  If the nerve goes to the pancreas then diabetes can develop.

Over time the scar tissue continues to contract and cause more problems. This can take 20 to 60 years. Post Polio Syndrome is really a symptom of progressive dehydration of the central nervous system followed by dehydration of the peripheral nerves.

This dehydration of the central nervous system can be reversed by stretching the spine, loosening up the adhesions in the meningeal system, and allowing the spinal fluid to flow properly again.

Advanced Central Nervous System Restoration will accomplish this in 24-48 treatments spaced out over a 6-12 month period with annual follow-ups.

yperbaric oxygen therapy (HBOT) is the inhalation of 100 percent oxygen inside a hyperbaric chamber that is pressurized to greater than 1 atmosphere (atm). HBOT causes both mechanical and physiologic effects by inducing a state of increased pressure and hyperoxia. HBOT is typically administered at 1 to 3 atm. While the duration of an HBOT session is typically 90 to 120 minutes, the duration, frequency, and cumulative number of sessions have not been standardized.

HBOT is administered in two primary ways, using a monoplace chamber or a multiplace chamber. The monoplace chamber is the less-costly option for initial setup and operation but provides less opportunity for patient interaction while in the chamber. Multiplace chambers allow medical personnel to work in the chamber and care for acute patients to some extent. The entire multiplace chamber is pressurized, so medical personnel may require a controlled decompression, depending on how long they were exposed to the hyperbaric air environment.

The purpose of this report is to provide a guide to the strengths and limitations of the evidence about the use of HBOT to treat patients who have brain injury, cerebral palsy, and stroke. Brain injury can be caused by an external physical force (also known as traumatic brain injury, or TBI); rapid acceleration or deceleration of the head; bleeding within or around the brain; lack of sufficient oxygen to the brain; or toxic substances passing through the blood-brain barrier. Brain injury results in temporary or permanent impairment of cognitive, emotional, and/or physical functioning. Cerebral palsy refers to a motor deficit that usually manifests itself by 2 years of age and is secondary to an abnormality of at least the part of the brain that relates to motor function. Stroke refers to a sudden interruption of the blood supply to the brain, usually caused by a blocked artery or a ruptured blood vessel, leading to an interruption of homeostasis of cells, and symptoms such as loss of speech and loss of motor function.

While these conditions have different etiologies, prognostic factors, and outcomes, they also have important similarities. Each condition represents a broad spectrum, from barely perceptible or mild disabilities to devastating ones. All three are characterized by acute and chronic phases and by changes over time in the type and degree of disability. Another similarity is that the outcome of conventional treatment is often unsatisfactory. For brain injury in particular, there is a strong sense that conventional treatment has made little impact on outcomes.

Predicting the outcome of brain injury, cerebral palsy, and stroke is difficult. Prognostic instruments, such as the Glasgow Coma Scale (GCS) for brain injury, are not precise enough to reliably predict an individual patient’s mortality and long-term functional status. Various prognostic criteria for the cerebral palsy patient’s function have been developed over the years. For example, if a patient is not sitting independently when placed by age 2, then one can predict with approximately 95 percent confidence that he/she never will be able to walk. However, it is not possible to predict precisely when an individual patient is likely to acquire a particular ability, such as smiling, recognizing other individuals, or saying or understanding a new word.

Mortality and morbidity from a stroke are related to older age, history of myocardial infarction, cardiac arrhythmias, diabetes mellitus, and the number of stroke deficits. Functional recovery is dependent on numerous variables, including age, neurologic deficit, comorbidities, psychosocial factors, educational level, vocational status, and characteristics of the stroke survivor’s environment.

The report focuses on the quality and consistency of studies reporting clinical outcomes of the use of HBOT in humans who have brain injury, cerebral palsy, or stroke. This information can be used to help providers counsel patients who use this therapy and to identify future research needs.

Contraindications of
Hyperbaric Oxygen TherapyNational Hyperbaric Oxygen Therapy Staff
Contraindications to HBOT are not extensive and often temporary. Communication with your HBOT physician about concerns is essential. Contraindications include: Pneumothorax unless treated with a Heimlich valve.

* Claustrophobia (fear of enclosed spaces, which is usually controllable.
* Acute upper respiratory infection or sinusitis.
* Emphysema, which can usually be treated at lower atmospheric pressure.
* Uncontrolled high fever.
* History of ear complications.
* Thoracic surgery or spontaneous pneumothorax.

Seizures at pressure have been documented at one per 10,000 compressions in children with Cerbral Palsy (CP), a rate identical with the occurrence in the overall hyperbaric therapy population. Even though seizures are a frequent symptom CP, most CP children with seizure who have been treated with HBOT have shown reduced seizure activity.

Patients with seizure disorders may have recurrence of seizures while at pressure during Hyperbaric Oxygen Therapy.

In China, HBOT is being used to treat seizures in children.